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.5 3. I have found this document useful? Yes No (2) 4. I have found this document useful? Yes No Please reply with comments and /or suggestions.Glucocorticoids mediate sex differences in c-fos induction by endotoxin: role of the hypothalamus and pituitary.
Studies were undertaken to compare hypothalamic and pituitary c-fos responses to LPS in female and male rats to investigate the role of sex hormones in mediating these responses. The results indicate that differences between the sexes do not result from differences in the number of responding neurons, or in the extent of the induction of the c-fos gene, but from the sensitivity of females to glucocorticoids. Corticosterone significantly blocked the induction of c-fos in the paraventricular (PVN) and supraoptic nuclei (SON) of male rats, but had no effect in females. Because the ventrolateral hypothalamus (VLH) is present in both sexes, but more intense c-fos staining was present in the female, c-fos was re-induced in the male after lesions of the VLH. Even after these lesions, c-fos was still inhibited in the PVN and SON of female rats. To investigate the role of female sex hormones, adult ovariectomized females were primed with 17 beta estradiol and progesterone 24 h before they were injected with LPS. Although estradiol alone had no effect on c-fos induction, progesterone significantly increased c-fos in all regions examined in males. In addition, progesterone alone induced c-fos in the PVN and SON of female rats. These results indicate that, within certain neuronal subpopulations of the hypothalamus and pituitary, sex differences in c-fos induction are mediated by sex hormones, and progesterone exerts its effects by itself or in conjunction with estradiol.In a bizarre incident in the city of Khatam, western Iran, a dog and a
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K. Yokoi, [*On Brin’s rank of $3$-manifolds*]{}, Algebr. Geom. Topol. [**7**]{} (2007), 2071–2108.
K. Yokoi, [*On the fundamental groups of $3$-manifolds*]{}, Proc. London Math. Soc. (3) [**100**]{} (2010), 1–36.
Apoptosis in the spinal cord white matter during the pathogenesis of multiple sclerosis.
Evidence of apoptosis in the normal-appearing white matter (NAWM) of the central nervous system (CNS) in autoimmune diseases has led to the suggestion that apoptosis may play a role in the pathogenesis of white matter (WM) lesions. To test this hypothesis, we examined the extent to which apoptotic cells are present in the NAWM of the spinal cord in patients with multiple sclerosis (MS). Tissue samples of the spinal cord were obtained from 10 patients with MS (CIS, n = 6; RRMS, n = 4) and 7 controls. We identified apoptotic cells by in situ end-labeling of DNA fragments, using the terminal deoxynucleotidyl transferase-mediated deoxyuridine triphosphate nick end-labeling (TUNEL) method. Apoptotic cells were identified in the NAWM of both the patients with MS and the controls. The numbers of apoptotic cells in MS patients were significantly higher
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